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TGF- signaling Fibrosis is driven by a complex interplay of pathological processes, including activation of TGF-, dysregulated chemokine expression, metabolic dysfunction, cellular senescence, mitochondrial and endoplasmic reticulum (ER) stress, autophagy, DNA damage, and apoptosis [20, 33, 36, 37]

One of the most crucial consequences of this fact is significant release of them to the environment

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Age-associated changes in oxidative stress and NAD + metabolism in human tissue

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